Artículo
Mitochondrial dysfunction and mitophagy activation in blood mononuclear cells of fibromyalgia patients: implications in the pathogenesis of the disease
Autor/es | Cordero, Mario D.
Miguel Rodríguez, Manuel de Moreno-Fernández, Ana María Carmona López, María Inés Garrido Maraver, Juan Cotán, David Gómez Izquierdo, Lourdes Bonal Pitz, Pedro Pablo Campa Valera, Francisco Bullon, Pedro Navas, Plácido Sánchez Alcázar, José Antonio |
Departamento | Universidad de Sevilla. Departamento de Estomatología Universidad de Sevilla. Departamento de Citología e Histología Normal y Patológica |
Fecha de publicación | 2010 |
Fecha de depósito | 2016-04-07 |
Resumen | Introduction: Fibromyalgia is a chronic pain syndrome with unknown etiology. Recent studies have shown some evidence demonstrating that oxidative stress may have a role in the pathophysiology of fibromyalgia. However, it ... Introduction: Fibromyalgia is a chronic pain syndrome with unknown etiology. Recent studies have shown some evidence demonstrating that oxidative stress may have a role in the pathophysiology of fibromyalgia. However, it is still not clear whether oxidative stress is the cause or the effect of the abnormalities documented in fibromyalgia. Furthermore, the role of mitochondria in the redox imbalance reported in fibromyalgia also is controversial. We undertook this study to investigate the role of mitochondrial dysfunction, oxidative stress, and mitophagy in fibromyalgia. |
Cita | Cordero, M.D., Miguel Rodríguez, M.d., Moreno-Fernández, A.M., Carmona López, I.M., Garrido Maraver, J., Cotán, D.,...,Sánchez Alcázar, J.A. (2010). Mitochondrial dysfunction and mitophagy activation in blood mononuclear cells of fibromyalgia patients: implications in the pathogenesis of the disease. |
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