Perfil del autor: Calvo Baltanás, Fernando
Datos institucionales
Nombre | Calvo Baltanás, Fernando |
Departamento | Fisiología Médica y Biofísica |
Área de conocimiento | Fisiología |
Categoría profesional | Profesor Contratado Doctor |
Correo electrónico | Solicitar |
Estadísticas
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Nº publicaciones
7
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Nº visitas
856
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Nº descargas
946
Publicaciones |
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Artículo
Critical Requirement of SOS1 for Development of BCR/ABL-Driven Chronic Myelogenous Leukemia
(MDPI, 2022)
We showed previously that the ABL-mediated phosphorylation of SOS1 promotes RAC activation and contributes to BCR-ABL ... |
Trabajo Fin de Grado
Propuesta de entrenamiento para paliar las consecuencias del tratamiento ante diferentes tipos de cáncer de mama y leucemia infantil.
(2022)
En este proyecto hemos llevado a cabo una revisión de las principales guías sobre ejercicio para el paciente ... |
Artículo
The Childhood-Onset Neurodegeneration with Cerebellar Atrophy (CONDCA) Disease Caused by AGTPBP1 Gene Mutations: The Purkinje Cell Degeneration Mouse as an Animal Model for the Study of this Human Disease
(MDPI, 2021)
Recent reports have identified rare, biallelic damaging variants of the AGTPBP1 gene that cause a novel and documented ... |
Artículo
SOS2 Comes to the Fore: Differential Functionalities in Physiology and Pathology
(MDPI, 2021)
The SOS family of Ras-GEFs encompasses two highly homologous and widely expressed members, SOS1 and SOS2. Despite their ... |
Artículo
Critical requirement of SOS1 RAS-GEF function for mitochondrial dynamics, metabolism, and redox homeostasis
(Nature Publishing Group, 2021)
SOS1 ablation causes specific defective phenotypes in MEFs including increased levels of intracellular ROS. We showed that ... |
Artículo
Functional Specificity of the Members of the Sos Family of Ras-GEF Activators: Novel Role of Sos2 in Control of Epidermal Stem Cell Homeostasis
(MDPI, 2021)
Prior reports showed the critical requirement of Sos1 for epithelial carcinogenesis, but the specific functionalities of ... |
Artículo
SOS GEFs in health and disease
(Elsevier, 2020)
SOS1 and SOS2 are the most universal and widely expressed family of guanine exchange factors (GEFs) capable or activating ... |