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dc.creatorAyala Gómez, Antonioes
dc.creatorVenero Recio, José Luises
dc.creatorCano, Josefinaes
dc.creatorMachado, Albertoes
dc.date.accessioned2022-03-16T09:38:06Z
dc.date.available2022-03-16T09:38:06Z
dc.date.issued2007
dc.identifier.citationAyala Gómez, A., Venero Recio, J.L., Cano, J. y Machado, A. (2007). Mitochondrial toxins and neurodegenerative diseases. Frontiers in Bioscience-Landmark, 12 (3), 986-1007.
dc.identifier.issn2768-6701 (impresa)es
dc.identifier.issn2768-6698 (electrónica)es
dc.identifier.urihttps://hdl.handle.net/11441/130858
dc.description.abstractThe selective loss of a particular subset of neurons is a common feature of neurodegenerative disorders. A failure in respiratory chain complex activities in mitochondria seems to be a causative factor. The aim of this review is to describe the most important toxins affecting the mitochondrial function, which could be involved in the incidence of some of these diseases: MPTP, rotenone and 3-nitropropionic (3-NPA).es
dc.formatapplication/pdfes
dc.format.extent22 p.es
dc.language.isoenges
dc.publisherIMR Presses
dc.relation.ispartofFrontiers in Bioscience-Landmark, 12 (3), 986-1007.
dc.rightsAttribution-NonCommercial-NoDerivatives 4.0 Internacional*
dc.rights.urihttp://creativecommons.org/licenses/by-nc-nd/4.0/*
dc.subjectParkinson´s Diseasees
dc.subjectHuntington´s diseasees
dc.subjectRotenonees
dc.subject3-NPAes
dc.subjectIsoquinolineses
dc.subjectReviewes
dc.titleMitochondrial toxins and neurodegenerative diseaseses
dc.typeinfo:eu-repo/semantics/articlees
dcterms.identifierhttps://ror.org/03yxnpp24
dc.type.versioninfo:eu-repo/semantics/publishedVersiones
dc.rights.accessRightsinfo:eu-repo/semantics/openAccesses
dc.contributor.affiliationUniversidad de Sevilla. Departamento de Bioquímica y Biología Moleculares
dc.relation.publisherversionhttps://doi.org/10.2741/2119es
dc.identifier.doi10.2741/2119es
dc.journaltitleFrontiers in Bioscience-Landmarkes
dc.publication.volumen12es
dc.publication.issue3es
dc.publication.initialPage986es
dc.publication.endPage1007es

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