Author profile: Rodríguez Gómez, José Antonio
Institutional data
Name | Rodríguez Gómez, José Antonio |
Department | Bioquímica y Biología Molecular |
Knowledge area | Bioquímica y Biología Molecular |
Professional category | Profesor Titular de Universidad |
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Statistics
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No. publications
8
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No. visits
853
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No. downloads
820
Publications |
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Article
Selective deletion of Caspase-3 gene in the dopaminergic system exhibits autistic-like behaviour
(Elsevier, 2021)
Apoptotic caspases are thought to play critical roles in elimination of excessive and non-functional synapses and removal ... |
Article
Microglia: Agents of the CNS Pro-Inflammatory Response
(Multidisciplinary Digital Publishing Institute (MDPI), 2020)
The pro-inflammatory immune response driven by microglia is a key contributor to the pathogenesis of several neurodegenerative ... |
Article
Hydroxytyrosol Decreases LPS- and α-Synuclein-Induced Microglial Activation In Vitro
(MDPI, 2020)
Neuroinflammation is a common feature shared by neurodegenerative disorders, such as ... |
Article
Reformulating Pro-Oxidant Microglia in Neurodegeneration
(MDPI, 2019)
In neurodegenerative diseases, microglia-mediated neuroinflammation and oxidative stress are central events. Recent ... |
Article
Genetic Rescue of Mitochondrial and Skeletal Muscle Impairment in an Induced Pluripotent Stem Cells Model of Coenzyme Q10 Deficiency
(Wiley-Blackwell, 2017)
Coenzyme Q10 (CoQ10) plays a crucial role in mitochondria as an electron carrier within the mitochondrial respiratory chain ... |
Article
Selective accumulation of biotin in arterial chemoreceptors: requirement for carotid body exocytotic dopamine secretion
(2016)
Biotin is a water-soluble vitamin required for the function of carboxylases as well asfor the regulation of gene expression. ... |
Article
Potential use of adipose tissue stem cells in the control of aging
(Federation of Anti-Aging Medicine Societies, 2012)
Cell therapy with adult stem cells is a new battle front for the control of aging. Before being used for this purpose, we ... |
Article |