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Mitochondrial dysfunction and mitophagy activation in blood mononuclear cells of fibromyalgia patients: implications in the pathogenesis of the disease

Opened Access Mitochondrial dysfunction and mitophagy activation in blood mononuclear cells of fibromyalgia patients: implications in the pathogenesis of the disease

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Autor: Cordero, Mario D.
Miguel Rodríguez, Manuel de
Moreno-Fernández, Ana Maria
Carmona López, Inés M
Garrido Maraver, Juan
Cotán, David
Gómez Izquierdo, Lourdes
Bonal Pitz, Pedro Pablo
Campa Valera, Francisco
Bullon, Pedro
Navas, Plácido
Sánchez Alcázar, José Antonio
Departamento: Universidad de Sevilla. Departamento de Estomatología
Universidad de Sevilla. Departamento de Citología e Histología Normal y Patológica
Fecha: 2010
Tipo de documento: Artículo
Resumen: Introduction: Fibromyalgia is a chronic pain syndrome with unknown etiology. Recent studies have shown some evidence demonstrating that oxidative stress may have a role in the pathophysiology of fibromyalgia. However, it is still not clear whether oxidative stress is the cause or the effect of the abnormalities documented in fibromyalgia. Furthermore, the role of mitochondria in the redox imbalance reported in fibromyalgia also is controversial. We undertook this study to investigate the role of mitochondrial dysfunction, oxidative stress, and mitophagy in fibromyalgia.
Cita: Cordero, M.D., Miguel Rodríguez, M.d., Moreno-Fernández, A.M., Carmona López, I.M., Garrido Maraver, J., Cotán, D.,...,Sánchez Alcázar, J.A. (2010). Mitochondrial dysfunction and mitophagy activation in blood mononuclear cells of fibromyalgia patients: implications in the pathogenesis of the disease.
Tamaño: 2.346Mb
Formato: PDF

URI: http://hdl.handle.net/11441/39728

DOI: http://dx.doi.org/10.1186/ar2918

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