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dc.creatorRibera, Annaes
dc.creatorFernández-Cuenca, Felipees
dc.creatorBeceiro Casas, Alejandroes
dc.creatorBou, Germánes
dc.creatorMartínez-Martínez, Luises
dc.creatorPascual Hernández, Álvaroes
dc.creatorCisneros, José Migueles
dc.creatorRodríguez-Baño, Jesúses
dc.creatorPachón Díaz, Jerónimoes
dc.creatorVila, Jordies
dc.date.accessioned2016-05-16T14:42:24Z
dc.date.available2016-05-16T14:42:24Z
dc.date.issued2004-04
dc.identifier.issn0066-4804es
dc.identifier.urihttp://hdl.handle.net/11441/41284
dc.description.abstractAntimicrobial susceptibility was determined in 15 epidemiologically unrelated clinical isolates of Acinetobacter genospecies 3. Moreover, the mechanisms of resistance to some b-lactam antibiotics may be associated with the presence of a chromosomal cephalosporinase, AmpC, and the resistance to quinolones related to mutations in the gyrA and parC genes.es
dc.formatapplication/pdfes
dc.language.isoenges
dc.publisherAmerican Society for Microbiologyes
dc.relation.ispartofAntimicrobial Agents and Chemotherapy, 48(4), 1430–1432es
dc.rightsAttribution-NonCommercial-NoDerivatives 4.0 Internacional*
dc.rights.urihttp://creativecommons.org/licenses/by-nc-nd/4.0/*
dc.titleAntimicrobial Susceptibility and Mechanisms of Resistance to Quinolones and b-Lactams in Acinetobacter Genospecies 3es
dc.typeinfo:eu-repo/semantics/articlees
dcterms.identifierhttps://ror.org/03yxnpp24
dc.type.versioninfo:eu-repo/semantics/publishedVersiones
dc.rights.accessRightsinfo:eu-repo/semantics/openAccesses
dc.contributor.affiliationUniversidad de Sevilla. Departamento de Medicinaes
dc.contributor.affiliationUniversidad de Sevilla. Departamento de Microbiología
dc.relation.publisherversionhttp://dx.doi.org/10.1128/aac.48.4.1430-1432.2004es
dc.identifier.doihttp://dx.doi.org/10.1128/aac.48.4.1430-1432.2004es
idus.format.extent3 p.es
dc.identifier.idushttps://idus.us.es/xmlui/handle/11441/41284

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