Browsing by Author "Garrido Maraver, Juan"
Bullon, Pedro; Alcocer-Gómez, Elísabet; Carrión, Ángel M.; Marín-Aguilar, Fabiola; Garrido Maraver, Juan; Román Malo, Lourdes Victoria (Mary Ann Liebert, Inc, 2016)
Impairment in adenosine monophosphate-activated protein kinase (AMPK) activity and NOD-like receptor family, pyrin domain containing 3 (NLRP3) inflammasome activation are associated with several metabolic and inflammatory ...
Apoptotic microtubules delimit an active caspase free area in the cellular cortex during the execution phase of apoptosis. [Article]Mata Fernández, Mario de la; Sánchez Alcázar, José Antonio; Miguel Rodríguez, Manuel de; Cordero Morales, Mario David; Zaderenko Partida, Ana Paula; Ybot González, Patricia; Cotán Marín, David; Oropesa Ávila, Manuel; Garrido Maraver, Juan (2013)
Mitochondrial dysfunction and mitophagy activation in blood mononuclear cells of fibromyalgia patients: implications in the pathogenesis of the disease [Article]Cordero, Mario D.; Miguel Rodríguez, Manuel de; Moreno-Fernández, Ana María; Carmona López, María Inés; Garrido Maraver, Juan; Cotán, David; Gómez Izquierdo, Lourdes; Bonal Pitz, Pedro Pablo; Campa Valera, Francisco; Bullon, Pedro; Navas, Plácido; Sánchez Alcázar, José Antonio (BioMed Central, 2010)
Introduction: Fibromyalgia is a chronic pain syndrome with unknown etiology. Recent studies have shown some evidence demonstrating that oxidative stress may have a role in the pathophysiology of fibromyalgia. However, it ...
Pharmacological Chaperones and Coenzyme Q10 Treatment Improves Mutant β-Glucocerebrosidase Activity and Mitochondrial Function in Neuronopathic Forms of Gaucher Disease [Article]Mata, Mario de la; Cotán, David; Oropesa Ávila, Manuel; Garrido Maraver, Juan; Cordero Morales, Mario David; Ortiz Mellet, Carmen; Ybot González, Patricia; García Fernández, José Manuel (Nature Publishing Group, 2015)
Gaucher disease (GD) is caused by mutations in the GBA1 gene, which encodes lysosomal β-glucocerebrosidase. Homozygosity for the L444P mutation in GBA1 is associated with high risk of neurological manifestations which are ...